Robert A Cramer, PhD
Associate Professor of Microbiology and Immunology
Microbiology and Immunology
Lawrence University Appleton, WI - BA Biology 1999
Colorado State University, MSc - 2001
Colorado State University, PhD - 2004
Duke University Medical Center, Post-Doctoral Fellow 2007
Molecular and Cellular Biology Graduate Programs
Molecular Pathogenesis Program
74 College Street Vail Loading Dock
Hanover NH 03755
Office: Remsen 208
Fungi, Fungal pathogenesis, hypoxia, innate immunity, metabolism, host-pathogen interactions
National Institutes of Heath, NIAID
Burroughs Wellcome Fund Investigator in the Pathogenesis of Infectious Diseases
Characterizing the Pathogenic, Genomic, and Chemical Traits of <i>Aspergillus fischeri</i>, a Close Relative of the Major Human Fungal Pathogen <i>Aspergillus fumigatus</i>.
Women receiving massive transfusion due to postpartum hemorrhage: A comparison over time between two nationwide cohort studies.
Protein Kinase A and High-Osmolarity Glycerol Response Pathways Cooperatively Control Cell Wall Carbohydrate Mobilization in <i>Aspergillus fumigatus</i>.
Beta-glucan-induced inflammatory monocytes mediate antitumor efficacy in the murine lung.
Response to Comment on "Sterilizing immunity in the lung relies on targeting fungal apoptosis-like programmed cell death".
Host-Derived Leukotriene B<sub>4</sub> Is Critical for Resistance against Invasive Pulmonary Aspergillosis.
Unique metabolic activation of adipose tissue macrophages in obesity promotes inflammatory responses.
Hyperbaric Oxygen Reduces Aspergillus fumigatus Proliferation <i>In Vitro</i> and Influences <i>In Vivo</i> Disease Outcomes.
Modulation of Immune Signaling and Metabolism Highlights Host and Fungal Transcriptional Responses in Mouse Models of Invasive Pulmonary Aspergillosis.
Overview of carbon and nitrogen catabolite metabolism in the virulence of human pathogenic fungi.