Caitlin G Howe, PhD
Assistant Professor of Epidemiology
Adjunct Research Assistant Professor (University of Southern California)
Columbia University (PhD), 2016
Pomona College (BA), 2009
Quantitative Biomedical Sciences
1 Medical Center Drive
Lebanon NH 03766
Dr. Howe’s research focuses on toxic metal exposures and their impacts on maternal and child health, with a particular interest in effects on early life growth and cardiometabolic health. Additional areas of interest include epigenetic mediators of toxicant exposures and metals toxicity in the context of nutritional status and complex environmental mixtures.
Prenatal Metal Mixtures, Fetal Growth, and the Role of MicroRNAs (NIEHS)
Professor Howe received her BA in 2009 from Pomona College, with a major in Biology and a minor in Spanish. After graduating, she worked at Fox Chase Cancer Center in an ovarian cancer research lab. She subsequently pursued a PhD in Environmental Health Sciences at Columbia University's Mailman School of Public Health. After receiving her PhD in 2016, she completed a postdoctoral research fellowship in the Department of Preventive Medicine at the University of Southern California. In 2020, she was appointed to the faculty at the Geisel School of Medicine.
Extracellular vesicle-enriched miRNA profiles across pregnancy in the MADRES cohort.
Exposure to metal mixtures in relation to blood pressure among children 5-7 years old: An observational study in Bangladesh.
Associations of metals and neurodevelopment: a review of recent evidence on susceptibility factors.
Extracellular vesicle microRNA in early versus late pregnancy with birth outcomes in the MADRES study.
Prenatal metal mixtures and child blood pressure in the Rhea mother-child cohort in Greece.
Prenatal Metal Mixtures and Birth Weight for Gestational Age in a Predominately Lower-Income Hispanic Pregnancy Cohort in Los Angeles.
Prenatal metal mixtures and fetal size in mid-pregnancy in the MADRES study.
Prenatal and postnatal mercury exposure and blood pressure in childhood.
Betaine and choline status modify the effects of folic acid and creatine supplementation on arsenic methylation in a randomized controlled trial of Bangladeshi adults.
Demographic predictors of urinary arsenic in a low-income predominantly Hispanic pregnancy cohort in Los Angeles.