Christopher H. Lowrey, MD
Title(s)
Professor of Medicine
Additional Titles/Positions/Affiliations
Chief, Division of Hematology
Vice Chair, Department of Medicine
Department(s)
Medicine
Education
Boston University School of Medicine, MD 1985
University of Pennsylvania, MA 1981
Bowdoin College, BA 1979
Programs
Dartmouth Cancer Center
Pharmacology and Toxicology Graduate Program
Program in Experimental and Molecular Medicine
Other
Websites
https:
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Contact Information
Christopher H. Lowrey, MD
Section of Hematology/Oncology
Dartmouth-Hitchcock Medical Center
Lebanon NH 03756
Office: 623 Rubin
Phone: 603-650-5747
Fax: 603-650-2334
Email: christopher.h.lowrey@dartmouth.edu
Assistant: Connie Goodrich
Asst. Phone: 603-650-2967
Asst. Email: Constance.M.Goodrich@hitchcock.org
Courses Taught
Medical Pharmacology (Cancer Chemotherapy) - DMS 2
Scientific Basis of Medicine (Hematology) - DMS 2
Program in Experimental and Molecular Medicine
Biography
Dr. Lowrey received his BA in Biochemistry from Bowdoin College in 1979 and his MA, also in Biochemistry, from the University of Pennsylvania in 1982. He received his MD from Boston University School of Medicine in 1985 and subsequently performed his residency in internal medicine at Tufts New England Medical Center in Boston and at the National Institutes of Health Clinical Center in Bethesda, MD. He then served as a Medical Staff Fellow and Senior Staff Fellow in the Clinical Hematology Branch of the National Heart, Lung, and Blood Institute of N.I.H. where he completed his fellowship in Hematology in 1992. In 1993 he joined the faculty of Dartmouth Medical School where he holds a joint appointment in the Departments of Medicine and Pharmacology.
Caregivers' Out-of-Pocket Expenses and Time Commitment Following Hematopoietic Stem Cell Transplantation at a Rural Cancer Center. Severe acquired platelet dysfunction because of primary myelofibrosis with full functional and morphological recovery after allogeneic hematopoietic cell transplantation. Hypoxia-Induced VISTA Promotes the Suppressive Function of Myeloid-Derived Suppressor Cells in the Tumor Microenvironment. Down the repressors! Up the fetal hemoglobin! Hospital readmission following transplantation: identifying risk factors and designing preventive measures. Induction of fetal hemoglobin through enhanced translation efficiency of γ-globin mRNA. Multiple physical stresses induce γ-globin gene expression and fetal hemoglobin production in erythroid cells. Laboratory tests for paroxysmal nocturnal hemoglobinuria. Vinblastine rapidly induces NOXA and acutely sensitizes primary chronic lymphocytic leukemia cells to ABT-737. Eukaryotic initiation factor 2α phosphorylation mediates fetal hemoglobin induction through a post-transcriptional mechanism. |
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