Brent L Berwin, Ph.D.
Associate Professor of Microbiology and Immunology
Microbiology and Immunology
University of Wisconsin-Madison, Ph.D., 1999
Lewis & Clark College, B.S., 1991
Dr. Berwin graduated from the University of Wisconsin-Madison in 1999. He did his postdoc training at Duke University and joined Dartmouth Medical School in the fall of 2004. Dr. Berwin is an Associate Professor in the Department of Microbiology and Immunology.
Molecular and Cellular Biology Graduate Programs
Norris Cotton Cancer Center
Dartmouth Medical Center
1 Medical Center Dr.
HB 7556 - Rm. 614W
Lebanon NH 03756
Monomethylarsonous Acid (MMAIII) Has an Adverse Effect on the Innate Immune Response of Human Bronchial Epithelial Cells to Pseudomonas aeruginosa.
Differential ASC requirements reveal a key role for neutrophils and a noncanonical IL-1β response to Pseudomonas aeruginosa.
Structure-based redesign of lysostaphin yields potent antistaphylococcal enzymes that evade immune cell surveillance.
Myeloid derived hypoxia inducible factor 1-alpha is required for protection against pulmonary Aspergillus fumigatus infection.
Acidosis potentiates the host proinflammatory interleukin-1β response to Pseudomonas aeruginosa infection.
CX3CR1 delineates temporally and functionally distinct subsets of myeloid-derived suppressor cells in a mouse model of ovarian cancer.
Pseudomonas aeruginosa flagellar motility activates the phagocyte PI3K/Akt pathway to induce phagocytic engulfment.
Mechanisms of phagocytosis and host clearance of Pseudomonas aeruginosa.
Acat1 gene ablation in mice increases hematopoietic progenitor cell proliferation in bone marrow and causes leukocytosis.
Flagellar motility is a key determinant of the magnitude of the inflammasome response to Pseudomonas aeruginosa.