Brent L Berwin, PhD
Associate Professor of Microbiology and Immunology
Microbiology and Immunology
University of Wisconsin-Madison, Ph.D., 1999
Lewis & Clark College, B.S., 1991
Dr. Berwin graduated from the University of Wisconsin-Madison in 1999. He did his postdoc training at Duke University and joined Dartmouth Medical School in the fall of 2004. Dr. Berwin is an Associate Professor in the Department of Microbiology and Immunology.
Molecular and Cellular Biology Graduate Programs
Norris Cotton Cancer Center
Dartmouth Medical Center
1 Medical Center Dr.
HB 7556 - Rm. 614W
Lebanon NH 03756
Lumacaftor (VX-809) restores the ability of CF macrophages to phagocytose and kill Pseudomonas aeruginosa.
The effect of loss of O-antigen ligase on phagocytic susceptibility of motile and non-motile Pseudomonas aeruginosa.
Acidosis Exacerbates in vivo IL-1-Dependent Inflammatory Responses and Neutrophil Recruitment During Pulmonary Pseudomonas aeruginosa Infection.
Editor's Highlight: Nlrp3 Is Required for Inflammatory Changes and Nigral Cell Loss Resulting From Chronic Intragastric Rotenone Exposure in Mice.
Acidosis increases the susceptibility of respiratory epithelial cells to <i>Pseudomonas aeruginosa</i>-induced cytotoxicity.
Blood clot detection using magnetic nanoparticles.
Monomethylarsonous Acid (MMAIII) Has an Adverse Effect on the Innate Immune Response of Human Bronchial Epithelial Cells to Pseudomonas aeruginosa.
Differential ASC requirements reveal a key role for neutrophils and a noncanonical IL-1β response to Pseudomonas aeruginosa.
Structure-based redesign of lysostaphin yields potent antistaphylococcal enzymes that evade immune cell surveillance.
Myeloid derived hypoxia inducible factor 1-alpha is required for protection against pulmonary Aspergillus fumigatus infection.